شنبه 1 اردیبهشت 1403
[Archive]
دوره 6، شماره 3 - ( 4-1387 )
جلد 6 شماره 3 صفحات 0-105 |
برگشت به فهرست نسخه ها
Download citation:
BibTeX | RIS | EndNote | Medlars | ProCite | Reference Manager | RefWorks
Send citation to:
BibTeX | RIS | EndNote | Medlars | ProCite | Reference Manager | RefWorks
Send citation to:
Aali B S, Narooi S. Spontaneous ovarian hyperstimulation syndrome in a patient presenting with missed abortion. IJRM 2008; 6 (3) :105-0
URL: http://ijrm.ir/article-1-105-fa.html
URL: http://ijrm.ir/article-1-105-fa.html
Spontaneous ovarian hyperstimulation syndrome in a patient presenting with missed abortion. International Journal of Reproductive BioMedicine. 1387; 6 (3) :105-0
چکیده: (2000 مشاهده)
Background: Spontaneous ovarian hyperstimulation syndrome rarely occurs during pregnancy and is usually associated with high levels of human chorionic gonadotropin, in conditions such as molar or multifetal pregnancies.
Case report: Here we report spontaneous ovarian hyperstimulation in a patient presenting with missed abortion at 16th week of gestation, when serum ß-subunit human chorionic gonadotropin level detected to be 400 milliunit per milliliter. Evacuation and curettage was performed and the ovaries returned to about normal size two months later.
Conclusion: Spontaneous ovarian hyperstimulation syndrome can develop even in the presence of very low levels of hCG in missed abortion
Case report: Here we report spontaneous ovarian hyperstimulation in a patient presenting with missed abortion at 16th week of gestation, when serum ß-subunit human chorionic gonadotropin level detected to be 400 milliunit per milliliter. Evacuation and curettage was performed and the ovaries returned to about normal size two months later.
Conclusion: Spontaneous ovarian hyperstimulation syndrome can develop even in the presence of very low levels of hCG in missed abortion
نوع مطالعه: Original Article |
فهرست منابع
1. Delbaere A, Smits G, Olatunbosun O, Pierson R, Vassart G, Costagliola S. New insights into the pathophysiology of ovarian hyperstimulation syndrome. What makes the difference between spontaneous and iatrogenic syndrome? Hum Reprod 2004; 19:486-489. [DOI:10.1093/humrep/deh124]
2. Michaelson-Cohen R, Altarescu G, Beller U, Reens R, Halevy-Shalem T, Eldar-Geva T. Does elevated human chorionic gonadotropin alone trigger spontaneous ovarian hyperstimulation syndrome? Fertil Steril 2007; [Epub ahead of print]. [DOI:10.1016/j.fertnstert.2007.09.049]
3. Check JH , Choe JK , Nazari A . Hyperreactio luteinalis despite the absence of a corpus luteum and suppressed follicle stimulating concentrations in a triplet pregnancy.Hum Reprod 2000; 15, 1043-1045. [DOI:10.1093/humrep/15.5.1043]
4. Ludwig M, Gembruch U, Bauer O , Diedrich K. Ovarian hyperstimulation syndrome (OHSS) in a spontaneous pregnancy with fetal and placental triploidy: information about the general pathophysiology of OHSS. Hum Reprod 1998; 13: 2082-2087. [DOI:10.1093/humrep/13.8.2082]
5. Arora R, Merhi ZO, Khulpateea N, Roth D, Minkoff H. Ovarian hyperstimulation syndrome after a molar pregnancy evacuation. Fertil Steril 2008; Epub ahead of print. [DOI:10.1016/j.fertnstert.2007.09.067]
6. De Leener A, Montanelli L, Van Durme J, Chae H, Smits G, Vassart G, et al. Presence and absence of FSH receptor mutations provide some insights to spontaneous ovarian hyperstimulation syndrome physiopathology. J Clin Endocrinol Metab 2006; 91:555-562. [DOI:10.1210/jc.2005-1580]
7. Nappi RG, Di Nero E, D'Aries AP, Nappi L. Natural pregnancy in hypothyroid woman complicated by spontaneous ovarian hyperstimulation syndrome. Am J Obstet Gynecol 1998; 178: 610-611. [DOI:10.1016/S0002-9378(98)70448-X]
8. Smits G, Olatunbosun OA, Delbaere A, Pierson RA, Vassart G, et al . Spontaneous ovarian hyperstimulation syndrome caused by a mutant follitropin receptor. N Engl J Med 2003; 349: 760-766. [DOI:10.1056/NEJMoa030064]
| بازنشر اطلاعات | |
 |
این مقاله تحت شرایط Creative Commons Attribution-NonCommercial 4.0 International License قابل بازنشر است. |
